Corpus Callosum
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Corpus Callosum
Corpus Callosum. The corpus callosum is the brain's largest fiber track pathway. It interconnects the two cerebral hemispheres and is divided, front to back, into four parts---the rostrum, the genu, the body, and the splenium. It is a fundamental structure, and its fiber tracts are essential to the interhemispheric exchange of brain signals, including the support of memory, learning, and sensory functions of the brain.
The corpus callosum coordinates signals from different parts of the brain and helps us in our thinking.
illustration of agenesis of the corpus callosum Agenesis of the corpus callosum is a birth defect in which this structure in the brain is either partially or completely missing. It may occur as an isolated brain problem, in combination with other brain abnormalities, such as ventriculomegaly, or in combination with problems with other organs, such as a congenital heart defect.
Agenesis of the corpus callosum is caused by disruption to development of the fetal brain. This disruption may be related to chromosome errors, genetic factors, prenatal infections, or other factors related to the prenatal environment. In most cases, it is not possible to know what specifically caused the
The corpus callosum coordinates signals from different parts of the brain and helps us in our thinking.
illustration of agenesis of the corpus callosum Agenesis of the corpus callosum is a birth defect in which this structure in the brain is either partially or completely missing. It may occur as an isolated brain problem, in combination with other brain abnormalities, such as ventriculomegaly, or in combination with problems with other organs, such as a congenital heart defect.
Agenesis of the corpus callosum is caused by disruption to development of the fetal brain. This disruption may be related to chromosome errors, genetic factors, prenatal infections, or other factors related to the prenatal environment. In most cases, it is not possible to know what specifically caused the
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تاريخ التسجيل : 23/11/2010
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